The activities of our clinician and basic neuroscience researchers at the University of Saskatchewan are focused on obtaining a comprehensive understanding of the development, structure and function of the nervous system. Our goal is to gain a better understanding of the causes of neurological disorders, cognitive dysfunction and nervous system disease mechanisms.
Our clinicians and basic researchers develop a dynamic synergy to bring “bench” research to the “bed side” with an ultimate goal to directly impact patient care and health outcomes. We are working to develop new treatment strategies for patients with several neurological diseases. Research is targeted to cellular and biological mechanisms of aging, Alzheimer’s disease, dementia, diabetes, epilepsy, head and spinal cord injury, mental health and psychiatric disorders, multiple sclerosis, pain, Parkinson’s disease and stroke.
The research laboratories of many of our neuroscience researchers are housed in the new Academic Health Sciences Building where we foster an interactive environment through shared space and facilities to enhance collaborative and interdisciplinary research. Our researchers utilize a range of techniques from behavioral testing, cell biology, electrophysiology, electron and multiphoton microscopy, brain imaging, molecular biology and the synchrotron light source located at the University of Saskatchewan.
You are welcome to explore our website and learn more about our researchers, trainees, research projects, news items and opportunities.
I would be happy to hear from you, so please feel free to contact me with your questions or comments.
Department of Anatomy and Cell Biology
Director of the Cameco MS Neuroscience Research Center
University of Saskatchewan
Date: March 30th, 2017
Place: Room GD-04 Health Sciences Building D-Wing
Time: 3:00 pm
Previous work in our lab has shown electrical stimulation (ES) to improve peripheral nerve regeneration and remyelination following a focal demyelination insult, the latter effect also associated with a dramatic impact of ES on macrophage polarization to a “pro-repair state”. We continue these investigations to gain more insight into the broad cellular and molecular events underlying these effects and now report how ES impacts activity-regulated-cytoskeletal (Arc) protein and CCL2 chemokine expression in the injured sciatic nerve.
Contact: Dr. Francisco S. Cayabyab, firstname.lastname@example.org, 306-966-8191